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I published widely and a monograph on Pott’s paraplegia (Oxford University Press generic cefadroxil 250mg with amex, 1956) 250 mg cefadroxil amex, written in collaboration with my old school-fellow Herbert Seddon and my Oswestry colleague Robert Roaf, led to my becoming hon- orary secretary of the Medical Research Council subcommittee on the treatment of spinal tubercu- losis. After Seddon’s retirement I became chair- man in 1974 and my duties involved regular travel in Africa and the Far East until 1981. David Lloyd GRIFFITHS I was fortunate to be a visiting professor, guest 1908–1997 lecturer or examiner in many countries, particu- larly in the Far East, and was the president’s guest I was born in 1908 in Wales, of Welsh parents, lecturer at the meeting of the American Orthope- and brought up as monoglot English, which I dic Association in 1972. In 1917 we moved I was fortunate to have no interest in or talent to Manchester, and I was educated at William for sport, and was able to devote my time to work, Hulme’s Grammar School, of which I eventually the Welsh language and literature, chamber music became Chairman of the Governors. I was delighted to become a member 121 Who’s Who in Orthopedics of the Court of the Royal National Eisteddfod of Wales. I was considered, by my friends, to be a good surgeon but enjoyed a vastly overrated repu- tation as a teacher. I was also a good after- dinner speaker, another rather valueless accomplishment. In 1939 I married Nancy Mary Webb, my dearly loved and unfailing supporter. David Lloyd Grifﬁths (Lloyd to his colleagues) was a remarkable person with his own ﬁrm views on matters orthopedic and general. He expressed these with clarity and honesty, sometimes with ascerbic intensity, but was a stickler for accuracy of expression. His “auto-obituary” infers that his career in Patrick HAGLUND Manchester left him unfulﬁlled, although one 1870–1937 must doubt that assessment. He certainly had a deep attachment to Oswestry as a senior member In its earliest stages Scandinavian orthopedic of the “Welsh ﬁrm” in harness with Sir Reginald practice developed largely along German lines. Watson-Jones, Rowland Hughes, Gruff Roberts, Special hospitals for the treatment of cripples Arwyn Evans and others.
In 1928 cefadroxil 250 mg without prescription, two Dutch engineers effective 250 mg cefadroxil, van der Pol and van der Mark, described the heartbeat by comparing it to a simple oscillator. This approach, which was revolution- ary at the time, gave rise to a whole family of models of the heartbeat and of the operation of other periodically active, electrically excitable cells (like neurones or skeletal muscle cells). This approach is, at the same time, the great advantage and a major limitation of membrane potential models. As they are rather compact, models of this type were the ﬁrst to be used in investigations of the spread of excitation in multi-dimensional ‘tissue’ representations consisting of relatively large numbers of interconnected excitable elements; their role in assessing biophysical behaviour like cardiac impulse propagation is undiminished. The major drawback of these models, however, is their lack of a clear reference between model components and constituent parts of the bio- logical system (e. These models, therefore, do not permit the simulation of patho-physiological detail, such as the series of events that follows a reduc- tion in oxygen supply to the cardiac muscle and, ultimately, causes serious disturbances in heart rhythm. A breakthrough in cell modelling occurred with the work of the British scientists, Sir Alan L. Their new electrical models calculated the changes in membrane potential on the basis of the underlying ionic currents. In contrast to the pre-existing models that merely portrayed membrane potentials, the new generation of models calculated the ion ﬂuxes that give rise to the changes in cell electrical potential. Thus, the new models pro- vided the core foundation for a mechanistic description of cell function. Since then, the study of cardiac cellular behaviour has made immense progress, as have the related ‘ionic’ mathematical models. There are various representations of all major cell types in the heart, descriptions of their metabolic activity, its relation to cell electrical and mechanical beha- viour, etc. Drug-receptor interactions and even the effects of modiﬁcations in the genetic information on cardiac ion channel-forming proteins have begun to be computed.